Gout: What are Tart Cherries?

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Post by Admin Wed Jun 08, 2016 8:56 am

Gout and What are Tart Cherries?

What are tart cherries? Other names: Prunus cerasus, sour cherry, quiche cherry, tart cherry juice, montmorency cherry, balaton cherry Cherries are the smallest members of the stone fruit family, which include plums, apricots, nectarines, and peaches.

Why do people use tart cherries and tart cherry juice? Both sweet and tart cherries and cherry juice have long been used by traditional healers as a folk remedy for gout, because cherries are thought to lower urate levels in the body.

Are Tart Cherries More Effective as Compared to Fairly Sweet Cherries?

Generally, tart cherries have been found to have higher concentrations of phenolics and antioch university los angeles cherries. After reading what was written here, don't you get the impression that you had actually heard about these points sometime back. Think back and think deeply about Gouty foods

Little randomized controlled trial in the British Journal of Sports Medicine examined the effectiveness of a sour cherry juice blend in preventing symptoms of exercise-induced muscle damage. Fourteen male college students drank 12 fl oz of a cherry juice blend or a placebo, 2 times per day for eight consecutive days. Power loss was significantly lower in people taking the cherry juice (4%) compared with the placebo (22%). Pain was also significantly lower in people taking the cherry juice. Jill M. Tall, Ph.D., hydrotherapy offers relief to dog with joint pain Hopkins, was the lead researcher of a study in which tested the effectiveness of orally administrated anthocyanins from tart cherries on inflammation-induced soreness in rats. The results of the study suggested that tart cherry anthocyanins may have a beneficial role in reducing inflammatory pain. One small study published in the Journal of Nutrition supported the anti-gout effectiveness of cherries. assessed the effects of Bing cherry (a sweet cherry) intake on healthy women and found that cherry intake decreased blood urate levels, and there was a minor decrease in inflammatory markers c-reactive protein and nitric oxide. Safety Evil or Very Mad

Test tube studies, cherry anthocyanins have been found to protect neurons from damage by oxidative stress. However, there have been no studies that have looked at regardless of whether cherry extracts may prevent or perhaps slow the progression of neurodegenerative conditions such as Alzheimer's disease or Parkinson's disease in humans. Embarassed

Cherries contain sorbitol, which may exacerbate symptoms in people with irritable bowel syndrome, small intestine microbial overgrowth, or fructose malabsorption.


Gout occurs when too much uric acid builds up in the blood and uric acid crystals precipitate in the cooler parts of the body including the joints of the hands or feet. High levels of uric acid may also build up as lumps under the skin known as tophi, or as kidney stones. Uric Acid is a waste product of the oxidation of purines which are constituents of nucleic acids such as Dna. Uric acid is normally excreted in the urine to keep a concentration of uric acid in the bloodstream of around 4 mg/dL. When the concentration exceeds 7 mg/dL, crystals of monosodium urate start to form in the tissues. This condition is known as hyperuricemia.


Allopurinol, probenecid, and colchicine are sometimes prescribed in daily doses to reduce the risk or lessen the severity of future episodes. Allopurinol blocks the formation of uric acid, probenecid decreases the regularity of assaults of gout by increasing the kidney's excretion of uric acid, albany college of pharmacy & health sciences swelling and help prevent the frequency of gout attacks. These drugs speed the elimination of uric acid from the body, and they also slow down the rate at which it is produced, nevertheless they may cause nausea or skin rashes as side effects.

How to stop gout attacks naturally? Because gout episodes are so painful, patients demand some kind of gout treatment, despite the fact that the treatments for gout are not very effective and have undesirable side effects. The most common treatments range from the administration of nonsteroidal anti-inflammatory drugs (NSAIDs) such as ibuprofen, indomethacin and naproxen. Aspirin is not used as it aggravates hyperuricemia by improving uric acid retention. 17 These types of drugs can cause stomach pain, bleeding and ulcers, and past a certain dosage, they do not provide additional relief.

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How is Gout Diagnosed? Gout is identified based on family history, specific signs, and laboratory tests. Diagnosis of gout is confirmed by the determination of high levels of uric acid in the blood, monosodium urate crystals in the fluid of an inflamed joint, more than one attack of severe arthritis, and the involvement of only one joint such as the foot, ankle, or knee. We consider that we have only touched the perimeter of information available on Gout. There is still a lot more to be learnt!


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